Page 413 - Critical Care Nursing Demystified
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398        CRITICAL CARE NURSING  DeMYSTIFIED



                     ANSWERS


                     CASE STUDY

                         1.   When you palpate the right forearm fistula site, you should feel a thrill (rushing of blood
                           pulsating under your fingers) and auscultate a bruit (swishing noise), which will deter-
                           mine patency. Do not forget to use a Doppler if you do not hear anything.
                         2.   Hyperkalemia is confirmed by tall, tented T waves; widening of the QRS; and flattening of
                           the P wave. Confirmation is done by the laboratory value of 7.2 mEq/L.
                         3.   Her findings are indicative of a patient in CRF who has not been dialyzed. She is retaining
                           sodium, potassium, and phosphate, which are seen as the kidneys cannot excrete these
                           and they cannot be dialyzed due to her clotting AV fistula. Since calcium is opposite of
                           phosphate in CRF, her levels are low, causing this level to rise.
                                ABGs indicate a metabolic acidosis because the pH is 7.25 with an HCO  level of less
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                           than 22. She is partially compensating for this as her low pCO  indicates she is blowing
                                                                              2
                           off her CO  (creating a respiratory alkalosis). But the acidosis is her primary problem as
                                    2
                           indicated by the pH, which will always tell you the primary acid-base disturbance. This is
                           confirmed by her respiratory rate in the 30s; she probably has Kussmaul’s respirations if
                           she is breathing fast and deep.

                               Her low Hgb and Hct tell you to ask the husband or patient if she has been taking her   Downloaded by [ Faculty of Nursing, Chiangmai University 5.62.158.117] at [07/18/16]. Copyright © McGraw-Hill Global Education Holdings, LLC. Not to be redistributed or modified in any way without permission.
                           Procrit, folic acid, and iron to help compensate for the lack of kidney production of native
                           erythropoietin.
                         4.   The priority treatment is to give this patient calcium gluconate IV to help stimulate the
                           heart to contract and prevent death by dysrhythmias (ventricular fibrillation or asystole).
                           A solution of glucose and insulin should be given to help drive the potassium back into
                           the cell. This can also reverse the hyperkalemia.
                                  She will need blood later due to her low Hct and Hbg. She cannot wait for Epogen
                            to work.
                         5.   Decreased cardiac output related to hyperkalemia as noted by ECG changes, hypoten-
                           sion, and dysrhythmias. Fluid volume excess related to lack of dialysis treatments as
                           manifested by elevated serum sodium and peripheral edema.
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