Page 183 - Critical Care Notes
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          ■ Note amount and color of feces. Hematest stool prn. Monitor CBC, PT, PTT,
            and blood chemistries.
          ■ Insert Foley catheter. Monitor intake and output. Assess fluid and elec-
            trolyte balance.
          ■ Administer high-dose proton pump inhibitors to maintain gastric pH
            >6.0. Histamine antagonists are not recommended. Consider misoprostol
            (prostaglandin analog), anticholinergics, or mucosal protective agents.
          ■ Administer IV or intra-arterial vasopressin (Pitressin) with caution. Consider
            octreotide (Sandostatin), terlipressin, ornipressin, or somatostatin, especial-
            ly if varices suspected.
          ■ If coagulopathy is present (↑ PTT), administer vitamin K 10 mg IV and fresh
            frozen plasma.
          ■ Administer tranexamic acid (Cyklokapron) if excessive bleeding and
            decreased fibrinolysis.
          ■ A specific protocol of medications is ordered if patient is H. pylori positive.
          ■ Provide emotional support to patient and family. Relieve anxiety and pain.
          ■ Prepare patient for possible endoscopic or surgical procedures:
            ■ Laser phototherapy
            ■ Endoscopic thermal or injection therapy
            ■ Intra-arterial embolization
            ■ Vagotomy, pyloroplasty, or total or partial gastrectomy
                            Complications
          ■ Gastric perforation → sudden and severe generalized abdominal pain with
            rebound tenderness and board-like abdominal rigidity
          ■ Reduced cardiac output, including hypovolemic shock
          ■ Nausea, vomiting, and diarrhea; pulmonary aspiration
          ■ Altered nutritional status with nutritional deficits; aspiration
          ■ Infection; fever, ↑ WBC and ↑ HR; sepsis
                        Esophageal Varices
          Esophageal varices are dilated, distended, tortuous veins in the esophagus.
          They may also occur in the proximal stomach. These varices most commonly
          result from portal hypertension (>10 mm Hg) secondary to hepatic cirrhosis
          caused by the consumption of large amounts of alcohol. Severe liver disease →
          blood coagulation abnormalities → bleeding.





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