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246  n  HyperTeNSION



           on  prevention,  Detection,  evaluation,  and   the United States, improvements since 1988
           Treatment  of  High  Blood  pressure  (JNc  7)   in awareness (81% vs. 69%), treatment (73%
   H       define HTN as two or more Bp readings of   vs. 54%), and control (50% vs. 27%) of HTN
           systolic  Bp  ≥140  mmHg  or  diastolic  Bp  ≥90   have  helped  to  recently  attain  the  Healthy
           mmHg  (chobanian  et  al.,  2003).  pre-HTN,   People 2010 goal of 50% control of HTN (egan,
           defined as systolic Bp ≥120 mmHg or diastolic   Zhao,  &  Axon,  2010).  Despite  the  improve-
           Bp ≥80 mmHg, increases the risk (up to twice   ments in HTN control over the last decade,
           the  risk)  of  developing  HTN  (chobanian   dramatic  disparities  in  the  prevalence  and
           et al., 2003). HTN is classified as either pri-  control of HTN exist in certain subpopula-
           mary HTN (formerly called essential HTN) or   tions (Institute of Medicine, 2003). For exam-
           secondary HTN. The cause of primary HTN,   ple, Blacks have higher rates of HTN, (40% vs.
           which  accounts  for  95%  of  cases,  remains   27 and 25% in Whites and Hispanics, respec-
           in question, but it is known to be correlated   tively;  Glover,  Greenlund,  Ayala,  &  croft,
           with  obesity,  increasing  age,  diabetes,  alco-  2005), higher average Bp, and more frequent
           hol consumption, and salt intake (carretero   target organ damage than other racial groups
           & Oparil, 2000). Secondary HTN accounts for   (lloyd-Jones et al., 2010). Mexican Americans
           the rest of the cases and results from identifi-  have disproportionately poor rates of HTN
           able disorders, such as chronic renal disease,   control  when  compared  with  other  groups
           renovascular  disease,  primary  aldosteron-  (17%  vs.  30%  in  both  Blacks  and  Whites)
           ism,  or  sleep  apnea,  and  may  resolve  with   despite comparable prevalence (Glover et al.,
           appropriate treatment of the underlying con-  2005).  Socioeconomic  status  is  consistently
           dition (chobanian et al., 2003).         inversely related to HTN prevalence, regard-
              HTN is a major risk factor for cardiovas-  less of race or ethnicity (Kaplan & Keil, 1993;
           cular disease, independent of other risk fac-  Mensah, Mokdad, Ford, Greenlund, & croft,
           tors, although it is related to other metabolic   2005).  Finally,  geographic  disparities  in
           risk  factors.  Metabolic  syndrome,  which  is   HTN control may contribute to higher rates
           increasing  in  prevalence,  comprises  a  con-  of stroke in the Southeastern United States
           stellation  of  risk  factors,  including  HTN,   than in other regions (Howard et al., 2006).
           abdominal obesity, dyslipidemia and insulin   These  disparities  are  most  likely  the  prod-
           resistance (chobanian et al., 2003). Although   uct of complex social, financial, and political
           causal origins are not well understood, met-  processes that result in barriers to effective
           abolic syndrome itself is a risk factor for the   health care and barriers to adoption of low-
           development  of  HTN,  and  the  presence  of   risk  lifestyles  (cooper  et  al.,  2000;  Institute
           HTN  alongside  metabolic  syndrome  poses   of  Medicine,  2003).  Unfortunately,  despite
           increased cardiovascular risk. Similar under-  recent attention to health disparities, dispar-
           lying  modifiable  lifestyle  risk  factors  have   ities in HTN in the United States have either
           been  identified  for  both  the  metabolic  syn-  persisted  or  worsened  in  the  past  three
           drome  and  HTN.  physical  inactivity,  diets   decades, continuing to place an undue bur-
           high in fats and refined carbohydrates, and   den  of  cardiovascular  risk  on  certain  sub-
           obesity are each associated with both HTN   groups of the population (cooper et al., 2000;
           and the metabolic syndrome. lifestyle modi-  Mensah et al., 2005).
           fications  of  these  risk  factors  and  smoking   The  current  approach  to  HTN  treat-
           cessation is a central to management of both   ment  relies  on  adherence  to  treatment
           disorders (Grundy et al., 2005).         algorithms,  such  as  the  recommendations
              Because  HTN  cannot  be  cured  in  the   of  JNc  7,  within  an  organized  health  care
           vast  majority  of  cases,  actions  to  increase   system  that  provides  regular  assessments
           awareness,  treatment  and  control  of  HTN   and  reviews  of  care  (Glynn,  Murphy,
           are critical to avert target organ damage. In   Smith,  Schroeder,  &  Fahey,  2010).  The  JNc
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