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                  108    PA R T  I I / Physiologic and Pathologic Responses
                   74. Berg, K., Dahlen, G., & Borresen, A. L. (1979). Lp(a) phenotypes, other  97. Mitchell, B. D., Blangero, J., Comuzzie, A. G., et al. (1998). A paired
                      lipoprotein parameters, and a family history of coronary heart disease in  sibling analysis of the beta-3 adrenergic receptor and obesity in Mexican
                      middle-aged males. Clinical Genetics, 16(5), 347–352.  Americans. Journal of Clinical Investigations, 101(3), 584–587.
                                              6
                                              6
                   75. Blumenthal, S., Jesse, M. J., Hennekens, C. H., et al. (1975). Risk fac-  98. Nagase, T., Aoki, A., Yamamoto, M., et al. (1997). Lack of association
                      tors for coronary artery disease in children of affected families. Journal  between the Trp64 Arg mutation in the beta 3-adrenergic receptor gene
                      of Pediatrics, 87(6, Pt. 2), 1187–1192.            and obesity in Japanese men: A longitudinal analysis. The Journal of
                               7
                               7
                   76. Hamby, R. I. (1981). Hereditary aspects of coronary artery disease.  Clinical Endocrinology Metabolism, 82(4), 1284–1287.
                      American Heart Journal, 101(5), 639–649.         99. Ristow, M., Muller-Wieland, D., Pfeiffer, A., et al. (1998). Obesity as-
                   77. Rissanen, A. M., & Nikkila, E. A. (1977). Coronary artery disease and  sociated with a mutation in a genetic regulator of adipocyte differentia-
                      its risk factors in families of young men with angina pectoris and in con-  tion. New England Journal of Medicine, 339(14), 953–959.
                      trols. British Heart Journal, 39(8), 875–883.   100. Sina, M., Hinney, A., Ziegler, A., et al. (1999). Phenotypes in three pedi-
                   78. Rosengren, A., Wilhelmsen, L., Eriksson, E., et al. (1990). Lipoprotein  grees with autosomal dominant obesity caused by haploinsufficiency
                      (a) and coronary heart disease: A prospective case-control study in a  mutations in the melanocortin-4 receptor gene. American Journal of
                      general population sample of middle aged men. BMJ, 301(6763),  Human Genetics, 65(6), 1501–1507.
                      1248–1251.                                      101. Walder, K., Norman, R. A., Hanson, R. L., et al. (1998). Association be-
                   79. Anderson, A. J., Loeffler, R. F., Barboriak, J. J., et al. (1979). Occlusive  tween uncoupling protein polymorphisms (UCP2-UCP3) and energy
                      coronary artery disease and parental history of myocardial infarction.  metabolism/obesity in Pima Indians. Human Molecular Genetics, 7(9),
                                                                                                                   7
                                                                                                                   7
                      Preventive Medicine, 8(3), 419–428.                1431–1435.
                   80. Sharp, S. D., Williams, R. R., Hunt, S. C., et al. (1992). Coronary risk  102. Kraft, H. G., Lingenhel, A., Kochl, S., et al. (1996). Apolipoprotein(a)
                      factors and the severity of angiographic coronary artery disease in mem-  kringle IV repeat number predicts risk for coronary heart disease.
                                                                                                        6
                                                                                                        6
                      bers of high-risk pedigrees. American Heart Journal, 123(2), 279–285.  Arteriosclerosis, Thrombosis, and Vascular Biology, 16(6), 713–719.
                   81. Smithies, O., & Maeda, N. (1995). Gene targeting approaches to com-  103. Hixson, J. E. (1991). Apolipoprotein E polymorphisms affect athero-
                      plex genetic diseases: Atherosclerosis and essential hypertension. Pro-  sclerosis in young males. Pathobiological Determinants of Atherosclero-
                      ceedings of the National Academy of Science USA, 92(12), 5266–5272.  sis in Youth (PDAY) Research Group. Arteriosclerosis Thrombosis, 11(5),
                   82. Tamminen, M., Mottino, G., Qiao, J. H., et al. (1999). Ultrastructure  1237–1244.
                      of early lipid accumulation in ApoE-deficient mice. Arteriosclerosis,  104. Moore, J. H., Reilly, S. L., Ferrell, R. E., et al. (1997). The role of the
                      Thrombosis, and Vascular Biology, 19(4), 847–853.  apolipoprotein E polymorphism in the prediction of coronary artery dis-
                   83. Mehrabian, M., & Lusis, A. J. (1992). Molecular genetics of coronary ar-  ease age of onset. Clinical Genetics, 51(1), 22–25.
                      tery disease. Candidate genes and processes in atherosclerosis. New York:  105. Wang, X. L., McCredie, R. M., & Wilcken, D. E. (1995). Polymor-
                      Karger.                                            phisms of the apolipoprotein E gene and severity of coronary artery
                   84. Heeneman, S., Lutgens, E., Schapira, K. B., et al. (2008). Control of  disease defined by angiography. Arteriosclerosis, Thrombosis, and Vascular
                      atherosclerotic plaque vulnerability: Insights from transgenic mice. Fron-  Biology, 15(8), 1030–1034.
                      tiers in Bioscience, 13, 6289–6313.             106. Wilson, P. W., Schaefer, E. J., Larson, M. G., et al. (1996). Apolipopro-
                   85. Stoll, M., Kwitek-Black, A. E., Cowley, A. W., Jr., et al. (2000) New tar-  tein E alleles and risk of coronary disease. A meta-analysis. Arteriosclero-
                      get regions for human hypertension via comparative genomics. Genome  sis, Thrombosis, and Vascular Biology, 16(10), 1250–1255.
                                                                                                 6
                                                                                                 6
                      Research, 10(4), 473–482.                       107. Gudnason, V., Thormar, K., & Humphries, S. E. (1997). Interaction of
                   86. Villa-Colinayo, V., Shi, W., Araujo, J., et al. (2000). Genetics of athero-  the cholesteryl ester transfer protein I405V polymorphism with alcohol
                      sclerosis: The search for genes acting at the level of the vessel wall. Cur-  consumption in smoking and non-smoking healthy men, and the effect
                      rent Atherosclerosis Reports, 2(5), 380–389.       on plasma HDL cholesterol and apoAI concentration. Clinical Genetics,
                   87. Altshuler, D., Hirschhorn, J. N., Klannemark, M., et al. (2000). The  51(1), 15–21.
                      common PPARgamma Pro12Ala polymorphism is associated with de-  108. Kuivenhoven, J. A., Jukema, J. W., Zwinderman, A. H., et al. (1998).
                                                     6
                      creased risk of type 2 diabetes. Natural Genetics, 26(1), 76–80.  The role of a common variant of the cholesteryl ester transfer protein
                                                     6
                   88. Hart, L. M., Stolk, R. P., Dekker, J. M., et al. (1999). Prevalence of vari-  gene in the progression of coronary atherosclerosis. The Regression
                      ants in candidate genes for type 2 diabetes mellitus in The Netherlands:  Growth Evaluation Statin Study Group. New England Journal of Medi-
                      The Rotterdam study and the Hoorn study. The Journal of Clinical En-  cine, 338(2), 86–93.
                      docrinology and Metabolism, 84(3), 1002–1006.   109. Jukema, J. W., van Boven, A. J., Groenemeijer B, et al. (1996). The Asp9
                   89. Horikawa, Y., Oda, N., Cox, N. J., et al. (2000). Genetic variation in the  Asn mutation in the lipoprotein lipase gene is associated with increased
                      gene encoding calpain-10 is associated with type 2 diabetes mellitus.  progression of coronary atherosclerosis. REGRESS Study Group, In-
                                  6
                                  6
                      Natural Genetics, 26(2), 163–175.                  teruniversity Cardiology Institute, Utrecht, The Netherlands. Regression
                   90. Reis, A. F., Ye, W. Z., Dubois-Laforgue, D., et al. (2000). Association of  Growth Evaluation Statin Study. Circulation, 94(8), 1913–1918.
                      a variant in exon 31 of the sulfonylurea receptor 1 (SUR1) gene with  110. Sanghera, D. K., Aston, C. E., Saha, N., et al. (1998). DNA polymor-
                      type 2 diabetes mellitus in French Caucasians. Human Genetics, 107(2),  phisms in two paraoxonase genes (PON1 and PON2) are associated
                                                               7
                                                               7
                      138–144.                                           with the risk of coronary heart disease. American Journal of Human
                   91. Stone, L. M., Kahn, S. E., Fujimoto, W. Y., et al. (1996). A variation at  Genetics, 62(1), 36–44.
                      position -30 of the beta-cell glucokinase gene promoter is associated with  111. Gardemann, A., Stricker, J., Humme, J., et al. (1999). Angiotensinogen
                      reduced beta-cell function in middle-aged Japanese-American men. Di-  T174M and M235T gene polymorphisms are associated with the extent
                      abetes, 45(4), 422–428.                            of coronary atherosclerosis. Atherosclerosis, 145(2), 309–314.
                                                                         W
                                                                         W
                   92. Vinik, A., & Bell, G. (1988). Mutant insulin syndromes. Hormone and  112. Wang, J., Liu, Z., & Chen, B. (2000). Association between genetic
                      Metabolic Research, 20(1), 1–10.                   polymorphism of dopamine transporter  gene and susceptibility to
                   93. Frossard, P. M., Lestringant, G. G., Malloy, M. J., et al. (1999). Human  Parkinson’s disease. Zhonghua Yi Xue Za Zhi, 80(5), 346–348.
                      renin gene BglI dimorphism associated with hypertension in two inde-  113. Winkelmann, B. R., Russ, A. P., Nauck, M., et al.  (1999). An-
                                               6
                      pendent populations. Clinical Genetics, 56(6), 428–433.  giotensinogen M235T polymorphism is associated with plasma an-
                                               6
                   94. Williams, R. R., Hunt, S. C., Hopkins, P. N., et al. (1994). Evidence for  giotensinogen and cardiovascular disease. American Heart  Journal,
                      single gene contributions to hypertension and lipid disturbances: Defin-  137(4, Pt. 1), 698–705.
                      ition, genetics, and clinical significance. Clinical Genetics, 46(1, Special  114. Wang, J. G., & Staessen, J. A. (2000). Genetic polymorphisms in the
                                                           6
                                                           6
                      No.), 80–87.                                       renin-angiotensin system: Relevance for susceptibility to cardiovascular
                   95. Heinonen, P., Koulu, M., Pesonen, U., et al. (1999). Identification of a  disease. European Journal of Pharmacology, 410(2/3), 289–302.
                      three-amino acid deletion in the alpha2B-adrenergic receptor that is as-  115. Tiret, L., Bonnardeaux, A., Poirier, O., et al. (1994). Synergistic effects
                      sociated with reduced basal metabolic rate in obese subjects. The Journal  of angiotensin-converting enzyme and angiotensin-II type 1 receptor
                      of Clinical Endocrinology Metabolism, 84(7), 2429–2433.  gene polymorphisms on risk of myocardial infarction.  Lancet,
                   96. Large, V., Hellstrom, L., Reynisdottir, S., et al. (1997). Human beta-2  344(8927), 910–913.
                      adrenoceptor gene polymorphisms are highly frequent in obesity and as-  116. Cambien, F., Poirier, O., Lecerf, L., et al. (1992). Deletion polymor-
                                                                                F
                                                                                F
                      sociate with altered adipocyte beta-2 adrenoceptor function. Journal of  phism in the gene for angiotensin-converting enzyme is a potent risk fac-
                      Clinical Investigations, 100(12), 3005–3013.       tor for myocardial infarction. Nature, 359(6396), 641–644.
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