Page 498 - ACCCN's Critical Care Nursing
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Neurological Alterations and Management  475



               Research vignette, Continued
               TBI,  for  which  intraparenchymal  pressure  tipped  catheters  were   In terms of study design and methodological implications, there
               most  commonly  used.  It  should  be  noted  that  since  then   were limitations relating to the missing elements in the data set.
               improvement  has  been  noted  in  an  Australasian  study  with  the   However,  this  resulted  in  minor  quantitative,  rather  than  major
                                         72
               SAFE  study  in  patients  with  TBI   demonstrating  higher  ICP   qualitative changes to the findings. Similar degrees of missing data
               monitoring rates more in line with the TBI guidelines, using ven-  were  reported  in  the  European  historical  controls  study,  which
               tricular catheters (~75%) and conversely lower mortality (24.56%)   emphasises the difficulties inherent in assessing the epidemiology
               overall and (29.24%) in severe TBI.                of TBI.





               Learning activities

               1.  What clinical signs are indicative of a fractured base of skull?   ●  What are the clinical signs of coma?
                  Are the injuries noted on CT focal or diffuse?     ●  Where does the source of coma localise in the brain?
               2.  Reading the Case Study, interpret Sam’s vital signs in relation   ●  Which complications of TBI might lead to coma?
                  to cerebral perfusion. Are management changes required?  ●  What  are  the  key  treatment  options  to  prevent  cerebral
               3.  Ischaemia  prevention  requires  a  PbtO 2 >20.  How  can  this  be   ischaemia?
                  achieved?                                       7.  A child is taken to the emergency room with lethargy, fever and
               4.  What is the pathophysiological basis for the rise in ICP? How   a stiff neck on examination.
                  would this manifest on the ICP waveform?           ●  What findings on initial lumbar puncture indicate bacterial
               5.  A  20-year-old  man  suffered  spinal  cord  injury  at  the  C2–C3   versus viral meningitis?
                  level  as  the  result  of  a  motorcycle  accident.  Explain  the   ●  In the case of bacterial meningitis, what are the most likely
                  effects  of  this  man’s  injury  on  ventilation  and  communica-  organisms?
                  tion; sensorimotor function; autonomic nervous system func-  8.  Your  patient  had  symptoms  of  an  ischaemic  stroke  approxi-
                  tion;  bowel,  bladder  and  sexual  function;  and  temperature   mately 2 hours ago and is undergoing a confirmatory CT scan
                  regulation.                                        in 30 minutes. You know tPA must be administered within 3
               6.  A 25-year-old-man is an unbelted driver involved in a motor   hours of the symptoms. What actions would you take? What is
                  vehicle accident and presents in a coma.           your rationale for these actions?





             ONLINE RESOURCES                                     National Stroke Foundation of Australia publication: Did you know that? http://
                                                                    www.strokefoundation.com.au
             American  Association  of  Spinal  Cord  Injury  Nurses  (AASCIN),http://  Neurological Foundation of New Zealand, http://www.neurological.org.nz/
               www.aascin.org                                     Post  Coma  Unresponsiveness  Guidelines,  http://www.nhmrc.gov.au/_files_
             The Brain Trauma Foundation, http://www.braintrauma.org  nhmrc/file/publications/synopses/e81.pdf
             Centers  for  Disease  Control:  Traumatic  Brain  Injury,  www.cdc.gov/  Spinal  Injury  Log  Roll  Protocol,  http://intensivecare.hsnet.nsw.gov.au/five/doc/
               traumaticbraininjuy/index.html                       logroll_guideline_R_cp_rnsh.pdf
             Cerebral  Spinal  Fluid  Drainage,  http://intensivecare.hsnet.nsw.gov.au/five/doc/  Spinal Injury Methylprednisolone Protocol, http://intensivecare.hsnet.nsw.gov.au/
               evd_csfspecimen_S_n_liverpool.pdf                    five/doc/methylprednisolone_spinalcord_D_svh.pdf
             Cervical  Collars,  http://intensivecare.hsnet.nsw.gov.au/five/doc/cervical_collars_  Stroke Foundation of New Zealand, http://www.stroke.org.nz/
               care_fitting_S_n_stgeorge.pdf                      Stroke   Management   Guidelines,   http://www.strokesociety.com.au/index.
             Australian Institute of Health and Welfare publication: Stroke, www.aihw.gov.au/  php?option=com_docman&Itemid=196
               cvd/stroke.cfm                                     Stroke  Thrombolytic  Protocol,  http://www.mja.com.au/public/issues/187_10_
             Australian & NZ Traumatic Brain Injury Study Results (ATBIS), www.anzics.com.au/  191107/bat11279_fm.pdf,
               ctg/completed-studies/50-atbis-  or  Brain  Injury  Association,  Inc.,  http://  Sports injuries: head and spine, www.injuryupdate.com.au/injuries/head_&_neck/
               www.biausa.org.                                      spinal_injuries.php
             Cervical   Traction,   http://intensivecare.hsnet.nsw.gov.au/five/doc/cervical_  Traumatic Brain Injury National Data Centre, http://www.tbindc.org/
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             Ethical guidelines for the care of people in post-coma unresponsiveness, http://
               www.nhmrc.gov.au/_files_nhmrc/file/publications/synopses/e81.pdf  REFERENCES
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               health.wa.gov.au/modelsofcare/docs/Stroke_Model_of_Care.pdf  4.  Strosznajder RP, Czubowicz K, Jesko H, Strosznajder JB. Poly(ADP-ribose)
             National Resource Centre for Traumatic Brain Injury, www.brainlink.org.au  metabolism in brain and its role in ischemia pathology. Mol Neurobiol 2010;
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