Page 837 - Williams Hematology ( PDFDrive )
P. 837

812            Part VI:  The Erythrocyte                                                                                                  Chapter 52:  Erythrocyte Disorders as a Result of Chemical and Physical Agents           813




                 22.  Celedon G, Gonzalez G, Barrientos D, et al: Stycholysin II, a cytolysin from the sea     59.  Vahter M, Counter SA, Laurell G, et al: Extensive lead exposure in children living in
                  anemone Stichodactyla helianthus promotes higher hemolysis in aged red blood cells.   an area with production of lead-glazed tiles in the Ecuadorian Andes. Int Arch Occup
                  Toxicon 51:1383, 2008.                                 Environ Health 70:282, 1997.
                 23.  Shen SC, Ham TH, Fleming EM: Studies on the destruction of red blood cells. III.     60.  Valentine WN, Paglia DE, Fink K, Madokoro G: Lead-poisoning: Association with
                  Mechanism and complications of hemoglobinuria in patients with thermal burns:   hemolytic-  anemia,  basophilic  stippling,  erythrocyte  pyrimidine  5′-nucleotidase
                  Spherocytosis and increased osmotic fragility of red blood cells. N Engl J Med 229:701,   deficiency, and intraerythrocytic accumulation of pyrimidines.  J Clin Invest 58:926,
                  1943.                                                  1976.
                 24.  Zarkowsky HS, Mohandas N, Speaker CB, Shohet SB: A congenital haemolytic-anemia     61.  Paglia DE, Valentine WN, Dahlgren JG: Effects of low-level lead-exposure on pyrim-
                  with thermal sensitivity of the erythrocyte membrane. Br J Haematol 29:537, 1975.  idine 5′-nucleotidase and other erythrocyte enzymes: Possible role of pyrimidine
                 25.  Prchal JT, Castleberry RP, Parmley RT, et al: Hereditary pyropoikilocytosis and ellipto-  5′-nucleotidase in pathogenesis of lead-induced anemia. J Clin Invest 56:1164, 1975.
                  cytosis: Clinical, laboratory, and ultrastructural features in infants and children. Pediatr     62.  Waldron HA: The anaemia of lead poisoning: A review. Br J Ind Med 23:83, 1966.
                  Res 16:484, 1982.                                     63.  Westerman  MP,  Pfitzer  E, Ellis  LD, Jensen WN:  Concentrations  of lead in  bone  in
                 26.  Schmid-Schonbein H, Wells R: Fluid drop-like transition of erythrocytes under shear.   plumbism. N Engl J Med 273:1246, 1965.
                  Science 165:288, 1969.                                64.  Lachant NA, Tomoda A, Tanaka KR: Inhibition of the pentose-phosphate shunt by
                 27.  Bull BS, Brailsford JD: Red-cell membrane deformability—New data.  Blood 48:663,   lead—A potential mechanism for hemolysis in lead-poisoning. Blood 63:518, 1984.
                  1976.                                                 65.  Khalil-Manesh F, Tartaglia-Erler J, Gonick HC: Experimental model of lead nephropa-
                 28.  Wagner HN, Gaertner RA, Feagin OT, et al: Removal of erythrocytes from circulation.   thy. IV. Correlation between renal functional changes and hematological indices of lead
                  Arch Intern Med 110:90, 1962.                          toxicity. J Trace Elem Electrolytes Health Dis 8:13, 1994.
                 29.  Taube H: Mechanisms of oxidation with oxygen. J Gen Physiol 49:29, 1965.    66.  Vincent PC: The effects of heavy metal ions on the human erythrocyte. I Comparisons
                 30.  Collman JP, Hermann PC, Fu L, et al: Aza-crown capped porphyrin models of myoglo-  of the action of several heavy metals. Aust J Exp Biol Med Sci 36:471, 1958.
                  bin: Studies of the steric interactions of gas binding. J Am Chem Soc 119:3481, 1997.    67.  Hasan J, Vihko V, Hernberg S: Deficient red cell membrane/Na  + K /-ATPase in lead
                                                                                                            +
                                                                                                               +
                 31.  Harris JW, Kellermeyer RW: The Red Cell: Production, Metabolism, Destruction: Normal   poisoning. Arch Environ Health 14:313, 1967.
                  and Abnormal, rev. ed. Harvard University Press, Cambridge, MA, 1970.    68.  Hernberg S, Nikkanen J: Enzyme inhibition by lead under normal urban conditions.
                 32.  Bunn HF, Forget BG:  Hemoglobin—Molecular, Genetic, and Clinical Aspects. WB     Lancet 1:63, 1970.
                  Saunders, Philadelphia, 1986.                         69.  Casado MF, Cecchini AL, Simao AN, et al: Free radical-mediated pre-hemolytic injury
                 33.  Niemoeller OM, Kiedaisch V, Dreischer P, et al: Stimulation of eryptosis by aluminium   in human red blood cells subjected to lead acetate as evaluated by chemiluminescence.
                  ions. Toxicol Appl Pharmacol 217:168, 2006.            Food Chem Toxicol 45:945, 2007.
                                              2+
                 34.  Sopjani M, Foller M, Lang F: Gold stimulates Ca  entry into and subsequent suicidal     70.  Brookfield RW: Blood changes occurring during the course of treatment of malignant
                  death of erythrocytes. Toxicology 244:271, 2008.       disease by lead, with special reference to punctate basophilia and the platelets. J Pathol
                 35.  Tavassoli M: Anemia of spaceflight. Blood 60:1059, 1982.  31:277, 1928.
                 36.  Mengel CE, Kann HE Jr, Heyman A, Metz E: Effects of in vivo hyperoxia on erythro-    71.  Clark M, Royal J, Seeler R: Interaction of iron deficiency and lead and the hematologic
                  cytes. II. Hemolysis in a human after exposure to oxygen under high pressure. Blood   findings in children with severe lead poisoning. Pediatrics 81:247, 1988.
                  25:822, 1965.                                         72.  White JM, Selhi HS: Lead and the red cell. Br J Haematol 30:133, 1975.
                 37.  Zimran A, Wasser G, Forman L, et al: Effect of ozone on red blood cell enzymes and     73.  Jensen WN, Moreno GD, Bessis MC: An electron microscopic description of basophilic
                  intermediates. Acta Haematol 102:148, 1999.            stippling in red cells. Blood 25:933, 1965.
                 38.  Biswas D, Banerjee M, Sen G, et al: Mechanism of erythrocyte death in human pop-    74.  Klein WJ Jr, Metz EN, Price AR: Acute copper intoxication. A hazard of hemodialysis.
                  ulation exposed to arsenic through drinking water.  Toxicol Appl Pharmacol 230:57,     Arch Intern Med 129:578, 1972.
                  2008.                                                 75.  Manzler AD, Schreiner AW: Copper-induced acute hemolytic anemia. A new compli-
                 39.  Mahmud H, Foller M, Lang F: Arsenic-induced suicidal erythrocyte death. Arch Toxicol   cation of hemodialysis. Ann Intern Med 73:409, 1970.
                  83:107, 2009.                                         76.  Deiss A, Lee GR, Cartwright GE: Hemolytic anemia in Wilson’s disease. Ann Intern
                 40.  Phoon WH, Chan MO, Goh CH, et al: Five cases of arsine poisoning. Ann Acad Med   Med 73:413, 1970.
                  Singapore 13:394, 1984.                               77.  Hansen PB: Wilson’s disease presenting with severe hemolytic anemia. Ugeskr Laeger
                 41.  Romeo L, Apostoli P, Kovacic M, et al: Acute arsine intoxication as a consequence of   150:1229, 1988.
                  metal burnishing operations. Am J Ind Med 32:211, 1997.    78.  McIntyre N, Clink HM, Levi AJ, et al: Hemolytic anemia in Wilson’s disease. N Engl J
                 42.  Rael LT, Ayala-Fierro F, Carter DE: The effects of sulfur, thiol, and thiol inhibitor com-  Med 276:439, 1967.
                  pounds on arsine-induced toxicity in the human erythrocyte membrane. Toxicol Sci     79.  Grudeva-Popova JG, Spasova MI, Chepileva KG, Zaprianov ZH: Acute hemolytic ane-
                  55:468, 2000.                                          mia as an initial clinical manifestation of Wilson’s disease. Folia Med (Plovdiv) 42:42,
                 43.  Winski SL, Barber DS, Rael LT, Carter DE: Sequence of toxic events in arsine-induced   2000.
                  hemolysis in vitro: Implications for the mechanism of toxicity in human erythrocytes.     80.  Fairbanks VF: Copper sulfate-induced hemolytic anemia. Inhibition of glucose-6-
                  Fundam Appl Toxicol 38:123, 1997.                      phosphate dehydrogenase and other possible etiologic mechanisms. Arch Intern Med
                 44.  Blair PC, Thompson MB, Bechtold M, et al: Evidence for oxidative damage to red blood   120:428, 1967.
                  cells in mice induced by arsine gas. Toxicology 63:25, 1990.    81.  Blume KG, Hoffbauer RW, Lohr GW, Rudiger HW: Genetische und biochemische
                 45.  Eysseric H, Vincent F, Peoc’h M, et al: A fatal case of chlorate poisoning: Confirmation   Aspekte der Pyruvatkinase menschlicher Erythrozyten. Verh Dtsch Ges Inn Med 75:450,
                  by ion chromatography of body fluids. J Forensic Sci 45:474, 2000.  1969.
                 46.  Caterson RJ, Savdie E, Raik E, et al: Heinz-body hemolysis in hemodialyzed patients     82.  Boulard M, Beutler E, Blume KG: Effect of copper on red-cell enzyme-activities. J Clin
                  caused by chloramines in Sydney tap water. Med J Aust 2:367, 1982.  Invest 51:459, 1972.
                 47.  Eaton JW, Kolpin CF, Swofford HS, et al: Chlorinated urban water—Cause of dialysis-     83.  Kiss JE, Berman D, Van Thiel D: Effective removal of copper by plasma exchange in
                  induced hemolytic-anemia. Science 181:463, 1973.       fulminant Wilson’s disease. Transfusion 38:327, 1998.
                 48.  Orringer EP, Mattern WD: Formaldehyde-induced hemolysis during chronic-hemodi-    84.  Stohlman F Jr, Brecher G, Schneiderman M, Cronkite EP: The hemolytic effect of ion-
                  alysis. N Engl J Med 294:1416, 1976.                   izing radiations and its relationship to the hemorrhagic phase of radiation injury. Blood
                 49.  Rice L, Alfrey CP: The negative regulation of red cell mass by neocytolysis: Physiologic   12:1061, 1957.
                  and pathophysiologic manifestations. Cell Physiol Biochem 15:245, 2005.    85.  Jin YS, Anderson G, Mintz PD: Effects of gamma irradiation on red cells from donors
                 50.  Risso A, Turello M, Biffoni F, Antonutto G: Red blood cell senescence and neocytolysis   with sickle cell trait. Transfusion 37:804, 1997.
                  in humans after high altitude acclimatization. Blood Cells Mol Dis 38:83, 2007.    86.  Lubash GD, Phillips RE, Bonsnes RW, Shields JD: Acute aniline poisoning treated by
                 51.  Klein M, Namer R, Harpur E, Corbin R: Earthenware containers as a source of fatal lead   hemodialysis—Report of case. Arch Intern Med 114:530, 1964.
                  poisoning—Case study and public-health considerations. N Engl J Med 283:669, 1970.    87.  Lowenstein L, Ballew DH: Fatal acute haemolytic anaemia, thrombocytopenic purpura,
                 52.  Andreasen NJ: Benjamin Franklin: Physicus et medicus. JAMA 236:57, 1976.  nephrosis and hepatitis resulting from ingestion of a compound containing apiol. Can
                 53.  Schwartz J, Landrigan PJ, Baker EL Jr, et al: Lead-induced anemia: Dose-response rela-  Med Assoc J 78:195, 1958.
                  tionships and evidence for a threshold. Am J Public Health 80:165, 1990.    88.  Schroder C, Kruger E, Abel J: [Acute poisoning caused by the herbicide dichlorprop
                 54.  Staudinger KC, Roth VS: Occupational lead poisoning. Am Fam Physician 57:719, 1998.  (preparation SYS 67 PROP)] [in German]. Kinderarztl Prax 59:81, 1991.
                 55.  Froom P, Kristal-Boneh E, Benbassat J, et al: Predictive value of determinations of zinc     89.  Martin H, Woerner W, Rittmeister B: Hemolytic anemia by inhalation of hydroxy-
                  protoporphyrin for increased blood lead concentrations. Clin Chem 44:1283, 1998.  lamines, with a contribution to the problem of Heinz body formation. Klin Wochenschr
                 56.  Autenrieth T, Schmidt T, Habscheid W: Lead poisoning caused by a Greek ceramic cup.   42:725, 1964.
                  Dtsch Med Wochenschr 123:353, 1998.                   90.  Fisher B: The significance of Heinz bodies in anemias of obscure etiology. Am J Med Sci
                 57.  Kakosy T, Hudak A, Naray M: Lead intoxication epidemic caused by ingestion of con-  230:143, 1955.
                  taminated ground paprika. J Toxicol Clin Toxicol 34:507, 1996.    91.  Nierenberg DW, Horowitz MB, Harris KM, James DH: Mineral spirits inhalation asso-
                 58.  Fischbein A, Wallace J, Sassa S, et al: Lead poisoning from art restoration and pottery   ciated with hemolysis, pulmonary edema, and ventricular fibrillation. Arch Intern Med
                  work: Unusual exposure source and household risk. J Environ Pathol Toxicol Oncol 11:7,   151:1437, 1991.
                  1992.                                                 92.  Hunter D: Industrial toxicology. Q J Med 12:185, 1943.








          Kaushansky_chapter 52_p0809-0814.indd   812                                                                   9/17/15   2:53 PM
   832   833   834   835   836   837   838   839   840   841   842