Page 303 - Color Atlas Of Pathophysiology (S Silbernagl Et Al, Thieme 2000)

A. Hyperinsulinism
Glucose

Oral
After antidiabetic
gastric resection drugs
Liver failure

Enzyme defect
Glucose
2 Hyperinsulinism, Hypoglycemia
1
Gastrointestinal Amino acids
hormones
K + Ca 2+
ATP In diabetic mother:
Beta cells Glucose Amino acids
of pancreas

3 In fetuses: Plate 9.19
Beta cell hyperplasia
Tumors
After birth:
Exogenous insulin supply increased 4
insulin release

Insulin-antagonistic
hormones Insulin
e.g. cortisol
5
Glucose consumption Lipolysis
by physical exercise, Hypoglycemia
tumors
Glucose formation Ketone bodies
by enzyme defects,
alcohol,
malnutrition 6
Abnormal supply to
Activation the nervous system
of sympathetic Voracious
nervous system appetite
Sweating Loss of
consciousness
Seizures
Tremors

Tachycardia Irreversible damage 293

Silbernagl/Lang, Color Atlas of Pathophysiology © 2000 Thieme
All rights reserved. Usage subject to terms and conditions of license.