Page 489 - Clinical Hematology_ Theory _ Procedures ( PDFDrive )

CHAPTER 23 ■ Myeloproliferative Neoplasms 473

REVIEW QUESTIONS (continued)

19. P tients with PRV e onstr te (n) o he osi - 26. A leukoerythrobl stic picture inclu es ll o the ollow-

erin in the bone rrow. ing except

A. bsence A. te r rop-sh pe erythrocytes

B. nor l ount B. nucle te erythrocytes

C. slightly incre se ount C. i ture ly phocytes

D. extre ely incre se ount D. i ture yeloi cells

20. re te p tients with PRV h ve li e expect ncy 27. Te e i n surviv l ti e or p tients with pri ry

er i gnosis. yelof brosis is pproxi tely ye r(s).

A. 1- to 6- onth A. 1

B. 6- to 12- onth B. 3

C. 1- to 5-ye r C. 5

D. ore th n 10-ye r D. 10

21. Te pri ry tre t ent or PRV is 28. I peripher l bloo s e r exhibits nu erous te r rop-

A. ther peutic phleboto y sh pe re bloo cells n bnor l pl telets, wh t is

B. yelosuppressive gents likely i gnosis?

C. r io ctive phosphorus A. He olytic ne i

D. low- ose busul n B. Iron ef ciency ne i

C. Myeloi et pl si

22. Pri ry yelof brosis is lso c lle D. Pernicious ne i

A. essenti l thro bocythe i

B. CML 29. T e le st co on or o MPN is

C. PRV A. PRV

D. gnogenic yeloi et pl si B. CML

C. pri ry yelof brosis

23. Te inci ence o pri ry yelof brosis is known to D. essenti l thro bocythe i

incre se er exposure to

A. sunshine 30. A jor criterion or the i gnosis o essenti l thro -

B. benzene bocythe i is

C. ntibiotics A. bsence o Ph chro oso e
1
D. inter eron B. incre se re bloo cell ss

C. il neutrophili in peripher l bloo

24. Te pre o in nt clinic l ni est tion o pri ry D. persistent incre se o pl telets in peripher l bloo

yelof brosis is

A. ne i 31. Te ost co on isor er in p tients with essenti l

B. spleno eg ly thro bocythe i is

C. e ull ry f brosis A. neurologic l ni est tions

D. ll o the bove B. thro botic or blee ing proble s

C. bnor l k ryotype

25. Te ost const nt e ture o pri ry yelof brosis is D. ne i

A. yserythropoiesis

B. ysleukopoiesis 32. Te bone rrow rchitecture in essenti l thro bocy-

C. ys eg k ryocytopoiesis the i is si il r to the rchitecture seen in

D. triline ge tur tion l isruption A. erythroi hyperpl si

B. leukocyte hyperpl si

C. CML

D. ly phocytic leuke i