Page 209 - Color Atlas Of Pathophysiology (S Silbernagl Et Al, Thieme 2000)

A. Causes and Consequences of Aortic Stenosis
0.5
4
Flow rate (L/s in systole) 0.3 0.6
Congenital 0.4 2 1.0 Aortic valve
opening
2
area (cm )
0.2
Calcifying 0.4
Acquired postnatally Left 0.1 0.2 (after Hurst)
Aorta atrium 0
Rheumatic P Ao 50 100 150 mmHg
inflammatory P LA Left Systolic pressure gradient
ventricle 1
Calcifying mm P LV – P Ao Stenosis
Hg ECG
P LV
150
Degenerative P LV Aortic
calcifying 100 P Ao
Aortic stenosis

50 a wave Plate 7.13
P LA (after Criley)
CO
0
(Click) Time
Systemic Ventricular
hypotension hypertension
II IV I SM II
2 Heart sound and murmur
Stimulation of
ventricular
baroreceptors (?)
Transmural
coronary artery Left heart
pressure hypertrophy
‘Paradoxical’
vasodilation Physical
exercise Coronary
blood flow Cardiac O 2
consumption

Myocardial hypoxia
(angina pectoris)
Vasodilation Arrhythmia
during exercise
Ventricular filling
Blood pressure
Left heart
Syncope failure 199

Silbernagl/Lang, Color Atlas of Pathophysiology © 2000 Thieme
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