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Evaluvation of Cardiac Syncope And ECG Markers of
                340
                                                     Sudden Cardiac Arrest.



              ly  in standing positions  or  after change  of posture
              suggest  vasovagal  etiology. Similarly  situations like
              micturition  cough/exercise  triggering  syncope  sug-
              gest  vasovagal  etiology.  Palpitations preceding  syn-
              cope or syncope post exercise  termination  suggest
              an arrhythmic cause. Syncope in lying down position
              mostly suggests arrhythmic/cardiac cause.
              High risk features in history are:

              Syncope post exercise or in supine position.
                                                                 Fig 1: Loss of R and deep Q waves.
              Palpitations at time of syncope
                                                                 2. Small voltage QRS complex indicate lack of elec-
              Family history of sudden cardiac arrest (SCA)      trically viable tissue  and splintered  QRS complexes
              History of structural heart disease or coronary artery   indicate a patchy scar.  Small  voltage  ECG may be
              disease (CAD)                                      suggestive of disorders like restrictive cardiomyopa-
                                                                 thy and amyloidosis.
              Past history of angioplasty or bypass surgery.

              Risk stratification on physical examination:
              High  risk  features suggesting  cardiac  etiology  on
              physical examination are:
              Heart rate less than 60 bpm or more than 150 bpm
              suggesting arrhythmic cause.

              Irregular heart rate suggest atrial fibrillation with vary-
              ing R-R interval.
              Low  blood  pressure  may suggest  tachycardia  with   Fig 2: Splintered QRS with low voltage.
              fast rates.
                                                                 3. Severe  LV  hypertrophy  is  suggestive  of  hypertro-
              Postural drop of BP suggests orthostatic intolerance.  phic heart disease.
              Chest examination may suggest cardiomegaly.
              Ejection systolic murmur is suggestive of aortic ste-
              nosis/ HOCM.
              S3 on examination may suggest severe LV dysfunc-
              tion.

              Risk stratification by ECG:
              Though patients of syncope may have normal ECGs,
              it is important to look for subtle changes which may
              suggest  arrhythmic problem  or  structural heart dis-
              ease. ECG changes would give a clue to cardiac eti-
              ology of syncope (2).
                                                                 Fig 3: LVH by voltage criteria: S wave in V2 + R wave
              A. ECG markers which may suggest                   in V5 > 35 mm
              structural heart disease:                          4. Increased  duration of  QRS complex  is  a marker

              1. Loss  of R waves and presence  of Q wave or  QS   for  delayed  intra ventricular  conduction  and if more
              pattern suggest  a transmural scar, evolved  infarct   than 150 ms is an independent predictor of VT/VF.
              which are substrate for arrhythmias.
                                                                 5.  ST  segment  and T  wave  changes: Persistent  ST
                                                                 elevation indicates aneurysm of LV or ongoing isch-
                                                                 emia. ST  depression  and/or bifid  or  deeply  inverted
                                                                 T  waves  suggests  ischemia of  myocardium. Spade


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