Page 492 - Cardiac Nursing
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                  468    P A R T  III / Assessment of Heart Disease
                  a derivation of Ohm’s law (pressure   flow   resistance), if re-  VENTRICULAR FUNCTION CURVES
                  sistance remains constant, there is a direct relationship between
                  pressure and flow. However, clinically, resistance is seldom con-  Knowledge of the relationship between preload, afterload, contrac-
                  stant. Thus, BP may appear adequate while flow is decreased, or  tility, and SV is essential for effective hemodynamic monitoring
                  conversely BP may be low although perfusion remains ade-  and guiding therapeutic actions that modify these hemodynamic
                  quate. Algorithms and evidence-based guidelines are available  variables. Ventricular function curves demonstrate the interaction
                                                                             V
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                  to guide assessment of the physiological and technical factors  between preload, afterload, and contractility and the effects of var-
                  that affect direct and indirect BP measurements and outline the  ious disease processes (heart failure [HF], hemorrhage) and thera-
                  correct performance and interpretation of these BP measure-  peutic actions (vasodilator or inotropic drug therapy) on SV and
                  ments. 22,23,97,98                                  CO (Fig. 21-8). The family of curves varies for each patient but is
                     In addition to the physical factors that cause the differences in  useful in predicting and evaluating the effects of various thera-
                  arterial pressure measured in various locations in the body, there  peutic interventions. The curves are constructed by plotting the
                  are also technical factors that affect measurement accuracy (see  PA occlusion pressure (PAOP) (or some measure of end-diastolic
                  Fig. 21-3). 23,99,100  In addition, the oscillometric system directly  volume or preload) on the horizontal axis and the CO, CI, or SV
                  measures the mean pressure and extrapolates the systolic and di-  on the vertical axis. A key point is that an increase in SV in re-
                  astolic pressure based on an algorithm, which may affect the ac-  sponse to a fluid bolus (change in preload) cannot be reliably pre-
                  curacy of the systolic and diastolic pressure measurements.  dicted on the basis of the standard preload indices (CVP, PAOP)
                     For direct and oscillometric BP monitoring the correct refer-  or volumetric indices (right ventricular [RV] end-diastolic vol-
                  ence is the heart. If the transducer or the arm is positioned above  ume, global end-diastolic volume), because the response depends
                  the heart, there will be a decrease in the measured pressure. Con-  on ventricular function, as indicated by the slope of the ventricu-
                  versely, if the transducer/arm is positioned below the heart, there  lar function curve. 117  The traditional preload indices remain use-
                  will be an increase in the measured pressure. 101–103  When the pa-  ful in the differential diagnosis and determining a patient’s risk for
                  tient is in the sitting position, for oscillometric or auscultated BP  pulmonary edema.
                  measurements, the arm should be supported at the level of the
                  heart (level of the midsternum). If the arm is parallel to the pa-
                  tient or supported on the armrest the SBP and DBP may be
                  10 mm Hg higher than if the arm is supported horizontally at heart
                  level (level of the midsternum) 104–109  and in patients with hyper-
                  tension the difference in arm position may cause a 20 mm Hg
                  overestimation of SBP. 108  If the patient is in a lateral recumbent
                  position, the noninvasive BP measurements taken from the “up
                  arm” may be 13 to 17 mm Hg lower than those if the patient is
                  in supine position, and BP measurements from the “down arm”  Cardiac Function  (CO, SV)
                  are similar to those taken at supine position or inconsis-
                  tent. 110–112  If the “up arm” is used, the measured pressure can be
                  corrected by measuring the distance from the angle-specific phle-
                  bostatic axis (see Display 21-1) and correcting the pressure (1 cm
                  0.73 mm Hg or 1 in.   1.8 mm Hg).
                     Forearm BP measurements may be necessary in cases in which
                  access to the upper arm is not possible or an appropriate cuff is  End-Diastolic Fiber Length
                  not available (i.e., the existence of morbid obesity or a conical-  (PA Occlusion Pressure)
                  shaped arm). 113  Two factors need to be considered when compar-  ■ Figure 21-8 Family of ventricular function curves representing
                  ing the BP from the upper arm and the forearm. First, if the arm  normal, depressed, and severely depressed function. A change in pre-
                  is in a dependent position, the hydrostatic pressure increases the  load is represented by a move up or down a single curve (Frank–Star-
                  BP in the forearm relative to the upper arm. To correct for this hy-  ling principle). Point A to point B and point B to point A reflect an
                  drostatic effect, the arm should be supported horizontally at heart  increase and decrease, respectively, in preload. The response to vol-
                                                                      ume loading is dependent on the position on the ventricular function
                  level. The second factor is that the SBP is normally higher in the  curve and the shape of the curve. If both ventricles are on the steep
                  periphery (forearm   upper arm), although there is limited re-  portion of the curve the SV will increase in response to volume (re-
                  search comparing noninvasive upper arm and forearm BP in he-  sponder). In contrast if the heart is on the flat portion of the curve the
                  modynamically stable patients. 114–116              SV will not increase (nonresponder). A change in afterload results in
                     A challenge when performing BP measurements in individ-  a shift in the curve that appears similar to that caused by contractil-
                  uals who are morbidly obese is finding an appropriately sized  ity, although the mechanism is different. Point D to E reflects the net
                  cuff. For every 5 cm increase in arm circumference (starting at  effect of a decrease in afterload on a failing heart. This upward and
                  35 cm) use of a standard cuff leads to an overestimation of SBP  lateral shift is the result of two actions. Point D to C reflects an in-
                  by 3 to 5 mm Hg and DBP by 1 to 3 mm Hg compared with  crease in force of contraction and point C to E a decrease in preload
                  an appropriately sized large cuff. 100  To correctly size the cuff,  due to increased systolic ejection. A change in contractility is repre-
                                                                      sented by an upward or downward shift of the curve, that is, for any
                  measure the arm circumference half the distance from the el-  given preload and afterload, the CO is increased or decreased. In a
                  bow to the wrist. Cuff size should be similar to the guidelines  failing heart, an additional effect of decreased contractility is an in-
                                         97
                  for upper arm circumference. The cuff should be centered be-  crease in preload due to decreased systolic ejection; thus, the net ef-
                  tween the elbow and wrist and the arm should be supported at  fect of a decrease in contractility is to shift the curve down and to the
                  the level of the heart.                             left (Point C to G).
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